hua wang - Toxicology

Author Information

Hua Wang is an established researcher affiliated with the Department of Toxicology at the School of Public Health, Anhui Medical University in China. Hua Wang is also associated with the Key Laboratory of Environmental Toxicology of Anhui Higher Education Institutes, China. The affiliation details were sourced from PubMed.

Research Contributions

Hua Wang has made notable contributions to the field of toxicology with a focus on environmental toxicants and their health impacts. Recent research includes studies on cadmium and its effects on the blood-testis barrier, arsenic-induced hepatic lipid accumulation, and selenium deficiency during gestation in relation to low birth weight. Other significant research areas include the impacts of cadmium exposure during pregnancy, oxidative stress, endoplasmic reticulum stress, and environmental metabolism disrupting chemicals. Hua Wang's work on fetal growth restriction, pulmonary fibrosis, and behavioral changes due to environmental exposures reflects a broad and impactful research portfolio.

Aliases

No aliases were provided or found for Hua Wang.

Publication and Citation Metrics

Metric	Value
Total Citation Count	5,070
h-index	44
Total Paper Count	167

Publications:

Environmental cadmium impairs blood-testis barrier via activating HRI-responsive mitochondrial stress in mice

DOI: 10.1016/j.scitotenv.2021.152247

Year: 2022

DNA hydroxymethylation reprogramming of β-oxidation genes mediates early-life arsenic-evoked hepatic lipid accumulation in adult mice

DOI: 10.1016/j.jhazmat.2022.128511

Year: 2022

Maternal selenium deficiency during gestation is positively associated with the risks for LBW and SGA newborns in a Chinese population

DOI: 10.1038/s41430-020-00809-x

Year: 2021

Gestational exposure to environmental cadmium induces placental apoptosis and fetal growth restriction via Parkin-modulated MCL-1 degradation.

DOI: 10.1016/j.jhazmat.2021.127268

Year: 2021

Environmental cadmium exposure during pregnancy causes diabetes-like phenotypes in mouse offspring: Association with oxidative stress in the fetal liver.

DOI: 10.1016/j.scitotenv.2021.146006

Year: 2021

Reactive oxygen species-evoked endoplasmic reticulum stress mediates 1-nitropyrene-induced epithelial-mesenchymal transition and pulmonary fibrosis.

DOI: 10.1016/j.envpol.2021.117134

Year: 2021

A review of environmental metabolism disrupting chemicals and effect biomarkers associating disease risks: Where exposomics meets metabolomics.

DOI: 10.1016/j.envint.2021.106941

Year: 2021

Paternal exposure to microcystin-LR induces fetal growth restriction partially through inhibiting cell proliferation and vascular development in placental labyrinth

DOI: 10.1007/s11356-021-14725-5

Year: 2021

Gestational arsenic exposure induces anxiety-like behaviors in adult offspring by reducing DNA hydroxymethylation in the developing brain.

DOI: 10.1016/j.ecoenv.2021.112901

Year: 2021

Serum CYR61 Is Associated With Airway Inflammation and Is a Potential Biomarker for Severity in Chronic Obstructive Pulmonary Disease

DOI: 10.3389/fmed.2021.781596

Year: 2021