piyajit watcharasit - Toxicology

Author Information

Piyajit Watcharasit is associated with the Laboratory of Pharmacology at the Chulabhorn Research Institute in Bangkok, Thailand. Additionally, Piyajit is affiliated with the Chulabhorn Graduate Institute and the Center of Excellence on Environmental Health and Toxicology (EHT), under the Office of the Higher Education Commission in Thailand. These affiliations highlight Piyajit's active involvement in research related to pharmacology and environmental health and toxicology.

Research Contributions

Piyajit Watcharasit has made significant contributions to the field of toxicology, with a focus on understanding the toxic effects and mechanisms of various substances, including arsenic and perfluorinated chemicals. Recent research by Piyajit includes investigating the impact of sodium arsenite on rat cortical astrocytes and its association with glycogen storage and toxicity, as well as exploring arsenic's disruption of neuronal insulin signaling. Other studies have delved into the anti-cancer potential of plant extracts and the effects of environmental toxins on cellular processes like apoptosis and inflammation. Piyajit's work is characterized by a strong emphasis on understanding the biochemical pathways and cellular responses to toxic exposures.

Aliases

The author is also known by the name "Piyajit Watcharasit".

Publication and Citation Metrics

Metric	Value
Total Citation Count	1471
H-index	19
Total Paper Count	44

Publications:

Increase of glycogen storage by sodium arsenite in rat cortical astrocytes through glycogen synthase activation and its association to toxicity

DOI: 10.1007/S13530-021-00094-6

Year: 2021

Arsenic disrupts neuronal insulin signaling through increasing free PI3K-p85 and decreasing PI3K activity.

DOI: 10.1016/j.toxlet.2021.06.002

Year: 2021

Anti‐cancer potentials of Gynura procumbens leaves extract against two canine mammary cancer cell lines

DOI: 10.1002/vms3.684

Year: 2021

Prolonged arsenic exposure increases tau phosphorylation in differentiated SH-SY5Y cells: The contribution of GSK3 and ERK1/2.

DOI: 10.1016/j.etap.2021.103626

Year: 2021

Sodium arsenite exposure impairs B cell proliferation and enhances vascular inflammation in Plasmodium berghei mouse model.

DOI: 10.1016/j.etap.2018.12.010

Year: 2019

Acute oral toxicity evaluation of Andrographis paniculata-standardized first true leaf ethanolic extract

DOI: 10.1016/j.toxrep.2019.05.003

Year: 2019

Arsenic impairs insulin signaling in differentiated neuroblastoma SH‐SY5Y cells

DOI: 10.1016/j.neuro.2018.03.004

Year: 2018

Genistein reduces the activation of AKT and EGFR, and the production of IL6 in cholangiocarcinoma cells involving estrogen and estrogen receptors.

DOI: 10.3892/ijo.2018.4375

Year: 2018

Paraquat induces extrinsic pathway of apoptosis in A549 cells by induction of DR5 and repression of anti-apoptotic proteins, DDX3 and GSK3 expression.

DOI: 10.1016/j.tiv.2017.04.016

Year: 2017

Perfluorinated chemicals, PFOS and PFOA, enhance the estrogenic effects of 17β‐estradiol in T47D human breast cancer cells

DOI: 10.1002/jat.3210

Year: 2016

Why is Fixation Important?

How Does Toxicity Manifest at the Organ Level?

What is the Role of the Liver in Detoxification?

How is Regulatory Policy Involved?

What Measures Can Be Taken to Protect Developing Bodies?

How Does GHS Address Environmental Concerns?

How does the FDA ensure the safety of dietary supplements?

How Can Drug-Induced Toxicity Be Detected?

What Role Does Risk Assessment Play in Microbial Safety?

What is the primary goal of rescue therapy in toxicology?

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