Richard Eugene Peterson - Toxicology

Author Information

Dr. Richard Eugene Peterson is a distinguished researcher affiliated with the School of Pharmacy and the Molecular and Environmental Toxicology Center at the University of Wisconsin-Madison. His extensive research is focused primarily on toxicology, with a specific interest in the impacts of various toxicants on biological systems. As a prominent figure in his field, Dr. Peterson’s contributions have significantly advanced our understanding of toxicological mechanisms and their implications on health.

Research Contributions

Dr. Peterson’s research portfolio is extensive and diverse, encompassing several critical areas within toxicology. His work includes the study of neuroanatomical mechanisms linking perinatal exposure to toxins like TCDD to urinary dysfunction in adulthood, the role of genetic background in susceptibility to hormone-induced urinary dysfunction, and the identification of biomarkers for glomerular injury. Additionally, his research also explores the synthesis and biological evaluation of compounds as agonists of the aryl hydrocarbon receptor. His investigations provide valuable insights into the non-malignant mechanisms of urinary dysfunction exacerbated by in utero and lactational exposure to environmental toxins.

Aliases

Dr. Richard Eugene Peterson is also known by various aliases including R E Peterson, Richard Peterson, Richard E Peterson, R. E. Peterson, and Richard E. Peterson.

Publication and Citation Metrics

Metric	Value
Total Citations	24,104
H-Index	75
Paper Count	305

Publications:

A neuroanatomical mechanism linking perinatal TCDD exposure to lower urinary tract dysfunction in adulthood.

DOI: 10.1242/dmm.049068

Year: 2021

A mechanism linking perinatal 2,3,7,8 tetrachlorodibenzo-p-dioxin exposure to lower urinary tract dysfunction in adulthood

DOI: 10.1242/dmm.049068

Year: 2021

Genetic background but not prostatic epithelial beta-catenin influences susceptibility of male mice to testosterone and estradiol-induced urinary dysfunction.

DOI:

Year: 2021

Symmetric Dimethylarginine Is a Sensitive Biomarker of Glomerular Injury in Rats

DOI: 10.1177/01926233211045341

Year: 2021

Synthesis and biological evaluation of FICZ analogues as agonists of aryl hydrocarbon receptor.

DOI: 10.1016/j.bmcl.2020.126959

Year: 2020

In utero and lactational 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) exposure exacerbates urinary dysfunction in hormone-treated C57BL/6J mice through a non-malignant mechanism involving proteomic changes in the prostate that differ from those elicited by testosterone and estradiol.

DOI:

Year: 2020

Impact of Sex, Androgens, and Prostate Size on C57BL/6J Mouse Urinary Physiology: Urethral Histology.

DOI: 10.1152/ajprenal.00540.2019

Year: 2020

Sox9 in mouse urogenital sinus epithelium mediates elongation of prostatic buds and expression of genes involved in epithelial cell migration.

DOI: 10.1016/j.gep.2019.119075

Year: 2019

Impact of Sex, Androgens, and Prostate Size on C57BL/6J Mouse Urinary Physiology: Functional Assessment.

DOI: 10.1152/ajprenal.00270.2019

Year: 2019

Insight and Resources From a Study of the “Impact of Sex, Androgens, and Prostate Size on C57BL/6J Mouse Urinary Physiology

DOI: 10.1177/0192623319877867

Year: 2019

What Types of Models are Used in Toxicology Simulations?

Why is Refinement Important?

What is Phylloquinone?

What Technologies are Used in Automated Imaging?

What are Anomalies in Toxicology?

What are the occupational hazards for workers in the fossil fuel industry?

Is Torrefaction Environmentally Friendly?

How Can We Reduce Variability-Related Risks?

What Are the Occupational and Environmental Exposures?

Why are Threshold Limits Important?

Partnered Content Networks

Relevant Topics