Toxins that target tubulin can cause a range of effects depending on their mechanism of action. These compounds can inhibit tubulin polymerization, destabilize microtubules, or promote excessive polymerization, all of which can disrupt essential cellular processes. For instance, colchicine, a well-known tubulin-binding toxin, prevents microtubule polymerization, leading to disrupted mitosis and inflammation control in gout.
