Genes play a crucial role in carcinogenicity, the process by which normal cells transform into cancer cells. Genetic mutations can activate oncogenes or inactivate tumor suppressor genes, leading to uncontrolled cell growth. Environmental carcinogens can cause DNA damage, leading to mutations. The body's ability to repair this damage is influenced by genetic factors, impacting the individual's risk of developing cancer. Understanding the genetic basis of carcinogenicity is essential for identifying at-risk populations and developing prevention strategies.
