Toxicological influences on hyperlipidemia occur through the interaction of chemicals with lipid metabolic pathways. Some toxicants can interfere with lipid synthesis, transport, and degradation. For instance, endocrine disruptors can mimic or inhibit natural hormones, leading to altered lipid homeostasis. Similarly, some medications, such as corticosteroids, beta-blockers, and thiazide diuretics, are known to increase lipid levels as a side effect.
